Search results for "Mental disorders"

showing 10 items of 1528 documents

P3‐039: Axonal neuritic pathology induces early presynaptic alterations in ps1/APP Alzheimer's mice hippocampus

2011

Loss of neurons in the hippocampus correlates with memory impairment in AD. Significant early reduction in the numerical density of hippocampal SOM interneurons was found in single (APPswe) and double (APPswe/ PS1dE9 and APPswe/TauP301S-G272V) transgenic models based on APP over expression and amyloid production. However, this inhibitory population was unaffected in age-matched single PS1 and tau transgenic mice as well as nontransgenic controls. Whereas SOM neuron loss in APPswe/PS1dE9 was associated to the onset of extracellular amyloid pathology in double APP/ tau mice this loss preceded plaque formation. Conclusions: As in human AD, somatostatin cell loss is a common early pathological …

Genetically modified mouseeducation.field_of_studyAmyloidEpidemiologyHealth PolicyTransgenePopulationHippocampusBiologyHippocampal formationInhibitory postsynaptic potentialPsychiatry and Mental healthCellular and Molecular NeuroscienceSomatostatinnervous systemDevelopmental Neurosciencemental disordersNeurology (clinical)Geriatrics and GerontologyeducationNeuroscienceAlzheimer's & Dementia
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Effects of l-Carnitine in Patients with Autism Spectrum Disorders: Review of Clinical Studies

2019

Carnitine is an amino acid derivative, which plays several important roles in human physiology, in the central nervous system, and for mitochondrial metabolism, in particular. Altered carnitine metabolic routes have been associated with a subgroup of patients with autism spectrum disorders (ASD) and could add to the pathophysiology associated with these disorders. We review the current evidence about the clinical effects of carnitine administration in ASD in both non-syndromic forms and ASD associated with genetic disorders. Two randomized clinical trials and one open-label prospective trial suggest that carnitine administration could be useful for treating symptoms in non-syndromic ASD. Th…

medicine.medical_specialtyAutism Spectrum DisorderCentral nervous systemPharmaceutical ScienceReviewComorbidityAnalytical Chemistrylaw.inventionlcsh:QD241-44103 medical and health sciences0302 clinical medicineNeurodevelopmental disorderlcsh:Organic chemistryRandomized controlled triallawCarnitineInternal medicinemental disordersDrug DiscoveryIntellectual disabilitymedicineHumansgenetic disordersGenetic Predisposition to DiseaseCarnitinePhysical and Theoretical ChemistryRandomized Controlled Trials as Topic030304 developmental biology0303 health sciencesDose-Response Relationship Drugbusiness.industryOrganic Chemistryvitaminmedicine.diseaseneurodevelopmental disorderPathophysiologyClinical trialTreatment Outcomemedicine.anatomical_structureChemistry (miscellaneous)Molecular MedicineAutismnutritional supplementationbusinessmetabolism030217 neurology & neurosurgerymedicine.drugMolecules
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REM sleep behavior disorder in a patient with frontotemporal dementia

2010

We describe a patient with frontotemporal dementia (FTD), a tauopathy, who also showed clinical and polysomnographic features of REM sleep behavior disorder (RBD). The patient is a 78-year-old male with a 1 year history of behavioral dysfunction involving emotion, character and social functioning. Brain imaging and the results of neuropsychological testing were consistent with a diagnosis of FTD. Sleep symptom onset occurred some years before the behavioral changes, and consisted of unpleasant dreams, vocalization, and prominent motor behaviors. A polysomnography confirmed the diagnosis of RBD. Our findings support the hypothesis that RBD, although more frequent in synucleinopathies, might …

Malemedicine.medical_specialtyNeurologyREM Sleep Behavior DisorderDermatologyPolysomnographyAudiologySettore BIO/09 - FisiologiaREM sleep behavior disorderMuscle tonemental disordersmedicineHumansPsychiatryAgedSleep disordermedicine.diagnostic_testElectromyographyREM sleep behaviorGeneral MedicineParasomniamedicine.diseaseElectrooculographyPsychiatry and Mental healthmedicine.anatomical_structureFrontotemporal DementiaSettore BIO/14 - FarmacologiaSettore MED/26 - NeurologiaNeurology (clinical)TauopathyPsychologyFrontotemporal dementiaNeurological Sciences
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Project DyAdd : Nonlinguistic theories of dyslexia predict intelligence

2020

Two themes have puzzled the research on developmental and learning disorders for decades. First, some of the risk and protective factors behind developmental challenges are suggested to be shared and some to be specific for a given condition. Second, language-based learning difficulties like dyslexia are suggested to result from or correlate with also nonlinguistic aspects of information processing. In the current study, we investigated how adults with developmental dyslexia and ADHD as well as healthy controls cluster across various dimensions designed to tap the prominent nonlinguistic theories of dyslexia. Participants were 18–55-year-old adults with dyslexia (n = 36), ADHD (n = 22), and…

procedural learningcomorbidityoppimisvaikeudetvisual attentionneuropsykologiakehityshäiriötdyslexiamental disordersADHDdysleksiaeyeblink conditioningtemporal processingvisual processing
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Biallelic mutations in neurofascin cause neurodevelopmental impairment and peripheral demyelination

2019

See Karakaya and Wirth (doi:10.1093/brain/awz273) for a scientific commentary on this article. Neurofascin (NFASC) isoforms are immunoglobulin cell adhesion molecules involved in node of Ranvier assembly. Efthymiou et al. identify biallelic NFASC variants in ten unrelated patients with a neurodevelopmental disorder characterized by variable degrees of central and peripheral involvement. Abnormal expression of Nfasc155 is accompanied by severe loss of myelinated fibres.

Male[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyNerve Fibers MyelinatedGene FrequencyNeurodevelopmental Disorder[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Nerve Growth FactorProtein IsoformsChildComputingMilieux_MISCELLANEOUSMyelin Sheathneurofascin; neurodevelopment; peripheral demyelinationAlleleneurodevelopmentDemyelinating DiseaseGenomicsneurodevelopment neurofascin peripheral demyelinationSettore MED/39 - Neuropsichiatria InfantilePedigree[SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyChild PreschoolPeripheral Nerve[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Femaleneurodevelopment; neurofascin; peripheral demyelinationNeurogliaHumanAdultAdolescentNervous System MalformationsGuillain-Barre SyndromeAxonNervous System MalformationneurofascinRanvier's NodesHumansNerve Growth FactorsPeripheral NervesAllelesAutoantibodiesperipheral demyelinationInfantProtein IsoformOriginal ArticlesAxonsnervous systemNeurodevelopmental DisordersCell Adhesion MoleculeMutationCell Adhesion MoleculesDemyelinating Diseases
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Disentangling the Amyloid Pathways: A Mechanistic Approach to Etiology

2020

Amyloids are fibrillar protein aggregates associated with diseases such as Alzheimer’s disease (AD), Parkinson’s disease (PD), type II diabetes and Creutzfeldt–Jakob disease. The process of amyloid polymerization involves three pathological protein transformations; from natively folded conformation to the cross-β conformation, from biophysically soluble to insoluble, and from biologically functional to non-functional. While amyloids share a similar cross-β conformation, the biophysical transformation can either take place spontaneously via a homogeneous nucleation mechanism (HON) or catalytically on an exogenous surface via a heterogeneous nucleation mechanism (HEN). Here, we postulate that…

prionnucleationmental disordersamyloidvirusAlzheiemr’sParkinson’slcsh:Neurosciences. Biological psychiatry. Neuropsychiatrylcsh:RC321-571Frontiers in Neuroscience
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The relationship between bipolar disorder and alcoholism: a controlled family study.

1995

SYNOPSISBipolar disorder and alcoholism are familial disorders. The familial–genetic relationship between both is controversial and has received insufficient study. This study explores whether bipolar disorder and alcoholism share familial risk factors, and whether the co-occurrence of lifetime diagnosis of bipolar disorder and alcoholism is familial. We report on first-degree relatives of 146 consecutively admitted patients with either bipolar disorder or/and alcoholism; relatives of the patients (in total 728 relatives directly interviewed) were compared with first-degree relatives of 109 general population probands (320 relatives directly interviewed). Overlap between the familial compon…

ProbandAdultMaleRiskmedicine.medical_specialtyBipolar DisorderAdolescentPopulationComorbidityGermanymental disordersmedicineHumansLife TablesBipolar disorderPsychiatryeducationApplied PsychologyAgededucation.field_of_studyDepressive DisorderModels GeneticFamilial riskMiddle Agedmedicine.diseaseComorbidityPsychiatry and Mental healthAlcoholismPhenotypeFemalePsychologyClinical psychologyPsychological medicine
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Pain-induced alterations in the dynorphinergic system within the mesocorticolimbic pathway: Implication for alcohol addiction.

2020

Latest studies have revealed that pain negatively impacts on reward processing and motivation leading to negative affective states and stress. These states not only reduce quality of life of patients by increasing the appearance of psychiatric comorbidities, but also have an important impact on vulnerability to drug abuse, including alcohol. In fact, clinical, epidemiological but also preclinical studies have revealed that the presence of pain is closely related to alcohol use disorders (AUDs). All this evidence suggests that pain is a factor that increases the risk of suffering AUD, predicting heavy drinking behavior and relapse drinking in those patients with a previous history of AUD. Th…

0301 basic medicinemedicine.medical_specialtyDopamineAlcohol use disorderMesolimbic pathwayκ-opioid receptor03 medical and health sciencesCellular and Molecular NeuroscienceReward system0302 clinical medicineQuality of lifeRewardmental disordersmedicineHumansPsychiatrybusiness.industryDopaminergicChronic painmedicine.diseaseSubstance abuseAlcoholism030104 developmental biologyQuality of LifeChronic Painbusiness030217 neurology & neurosurgeryJournal of neuroscience researchREFERENCES
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Project DyAdd: Non-linguistic Theories of Dyslexia Predict Intelligence

2020

Two themes have puzzled the research on developmental and learning disorders for decades. First, some of the risk and protective factors behind developmental challenges are suggested to be shared and some are suggested to be specific for a given condition. Second, language-based learning difficulties like dyslexia are suggested to result from or correlate with non-linguistic aspects of information processing as well. In the current study, we investigated how adults with developmental dyslexia or ADHD as well as healthy controls cluster across various dimensions designed to tap the prominent non-linguistic theories of dyslexia. Participants were 18–55-year-old adults with dyslexia (n = 36), …

VISUAL-ATTENTIONLANGUAGEBiological theories of dyslexiaProcedural memory3124 Neurology and psychiatryVisual processingprocedural learningBehavioral Neuroscience0302 clinical medicinePhysiopsychologie et psychologie biologique [psychiatrie]DEFICITSNeuropsychologieOriginal Research05 social sciencesInformation processingNeuropsychology16. Peace & justicePsychiatry and Mental healthcomorbidityNeuropsychology and Physiological PsychologyEyeblink conditioningNeurologyTEMPORAL-ORDERFATTY-ACIDSPsychologyCognitive psychology515 PsychologyBFeyeblink conditioningADULT DYSLEXIA050105 experimental psychologyCLASSIFICATIONlcsh:RC321-57103 medical and health sciencesWORKING-MEMORYBiopsychologie et psychopathologieNeurologiedyslexiamental disordersmedicineADHD0501 psychology and cognitive scienceslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological Psychiatryvisual processingPsychophysiologieWorking memoryDyslexia3112 NeurosciencesNeurosciences cognitivesPROCESSING ACUITYmedicine.diseasetemporal processingvisual attention13. Climate actionRC0321DEVELOPMENTAL DYSLEXIA030217 neurology & neurosurgeryNeurosciencePsychiatrie
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Revenue models, in-app purchase, and the app performance: Evidence from Apple's App Store and Google Play

2016

The effect of revenue models on app performance depends on the app store.The effect of in-app purchase on app performance depends on the app store.Paid, freemium and in-purchase models are shown to be effective in app store.Freemium and in-app purchase models are shown to be less effective in Google Play.App category influences the effects of both revenue models and in-app purchase. In this paper, we empirically examine how the revenue model (paid, free, or freemium) adopted for a given app affects the app revenue performance as measured by the app daily revenue rank. We also study the impact of in-app purchase on this measure of performance. Moreover, we study how such relationships are co…

ComputerSystemsOrganization_COMPUTERSYSTEMIMPLEMENTATIONGeneralLiterature_INTRODUCTORYANDSURVEYComputer Networks and CommunicationsE-commerceApp storeGeneralLiterature_MISCELLANEOUSManagement of Technology and Innovationmental disorders0502 economics and businessRevenueMobile app market Online distribution Revenue model E-commerce Empirical analysis.MarketingMarketingbusiness.industryInformationSystems_INFORMATIONSYSTEMSAPPLICATIONS05 social sciencesAdvertisingSettore ING-IND/35 - Ingegneria Economico-GestionaleFreemiumComputer Science ApplicationsLarge sampleRevenue modelFree model050211 marketingbusiness050203 business & management
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